Molekulare Medizin: Grundlagen, Pathomechanismen, by Matthias W. Hentze, Andreas E. Kulozik, C. R. Bartram,

By Matthias W. Hentze, Andreas E. Kulozik, C. R. Bartram, Christian Hagemeyer

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Extra info for Molekulare Medizin: Grundlagen, Pathomechanismen, Krankheitsbilder

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Note the biphasic responses ofNADH redox state and perfusion, in contrast to the steady decline in the forward rate constant of creatine kinase (Kf ) and flux of PCr to ATP during and after ACH infusion. HR x SBP =heart rate x systolic blood pressure; CO =cardiac output (IImin); MVOz =oxygen consumption (mllmin/l00 gm heart); PCr =phosphocreatine peak area; NADH =nicotinamide adenine dinucleotide (normalized to 100% Nz); Perfusion =ml/min/l00 gm eH NMR). crease. NADH fluorescence decreased and myocardial perfusion increased.

J Bioi Chern 241: 673-683, 1966 34 37. Schimerlik MI, Cleland WW: Inhibition of creatine kinase by chromium nucleotide. J BioI Chern 248: 8418-8423, 1973 38. Osbakken M, Mitchell MD, Zhang D, Mayevsky A, Chance B: In vivo correlation of myocardial metabolism, perfusion and mechanical function during increased cardiac work. Cardiovasc Res 25: 749-756, 1991 39. Osbakken M, Young M, Huddle J, Closter J, Prammer M, Chance B: Acute volume loading studied in cat myocardium with 31p nuclear magnetic resonance.

Circ Res 66: 135-146, 1990 Marban E, Koretsune Y, Corretti M, Chacko VP, Kusuoka H: Calcium and its role in myocardial cell injury during ischemia and reperfusion. Circulation 80(suppl IV): IVI7-IV22, 1989 Pierce GN, Maddaford TG, Kroeger EA, Cragoe EJ: Protection by benzamil against dysfunction and damage in rat myocardium after calcium depletion and repletion. Am J Physiol 258 (Heart Circ PhysioI27): HI7-H23, 1990 Ponce-Homos JE, Musi EA, Bonazzola P: Role of extracellular calcium on heart muscle energetics: effects of verapamil.

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